Human IL-19 ELISA Kit sinobiological KIT13089

$400.00
In stock
SKU
KIT13089
Catalog No.SizePrice (USD)
KIT13089-11 Kit (96 Tests)$414.71

General Information

Product nameHuman IL-19 ELISA Kit
SpecificityThis assay recognizes both recombinant and natural Human IL19.
ConjugateHRP
ShippingThis ELISA Kit is shipped at ambient temperature.
StorageUnopened Kit: Store at 2 - 8℃ Opened/Reconstituted Reagents: Please refer to CoA
Assay typeSolid Phase Sandwich ELISA (quantitative)
Sample typeSerum,Cell supernatant
Sensitivity37.85 pg/mL
Assay range62.5-4000 pg/mL
Materials provided1. 96 well microplate coated with Capture Antibody 2. Detection Antibody conjugated to HRP 3. Standards 4. Wash Buffer Concentrate 5. Dilution Buffer Concentrate 6. Color Reagent A 7. Color Reagent B 8. Stop Solution
Product overviewThis Human IL-19 ELISA Kit is an enzyme-linked immunosorbent assay for the quantitative measurement of Human IL-19 protein in Serum,Cell supernatant . It contains recombinant Human IL-19, and antibodies raised against the recombinant protein. This ELISA kit is complete and ready-to-use.

Synonyms: IL-10C ELISA Kit, Human; IL-19 ELISA Kit, Human; MDA1 ELISA Kit, Human; NG.1 ELISA Kit, Human; ZMDA1 ELISA Kit, Human

Image / Experimental Information

Image 1 DescriptionThis standard curve is only for demonstration purposes. A standard curve should be generated for each assay.
Image 1 Alt TextHuman IFNA5/IFNaG ELISA Kit Standard Curve 18800
Image 1 URLSource image
Image 2 DescriptionThis standard curve is only for demonstration purposes. A standard curve should be generated for each assay.
Image 2 Alt TextHuman IL19 / Interleukin-19 ELISA Kit Specificity 18803
Image 2 URLSource image

Background Information

Full Nameinterleukin 19
DescriptionThe molecular features at the IL19 locus may modestly alter the establishment of HIV-1 infection. Interleukin (IL) 19, IL-20, and IL-24 belong to the IL-10 cytokine family and have been identified to play a role in the regulation of epidermal functions and inflammation. The expression of IL19 in biopsies of patients with active ulcerative colitis was increased compared with patients with quiescent ulcerative colitis and that colitis was attenuated in IL-19-deficient mice. The disruption of the epithelial barrier with dextran sodium sulfate leads to increased IL-19 expression. Attenuated colitis in IL-19-deficient animals was associated with reduced numbers of IL-6-producing macrophages in the inflamed colonic lamina propria. Microbial-driven expression of IL-19 by intestinal macrophages may contribute to the pathogenesis of inflammatory bowel disease.

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